PRRT2 mutations are the major cause of benign familial infantile seizures.

نویسندگان

  • Julian Schubert
  • Roberta Paravidino
  • Felicitas Becker
  • Andrea Berger
  • Nerses Bebek
  • Amedeo Bianchi
  • Knut Brockmann
  • Giuseppe Capovilla
  • Bernardo Dalla Bernardina
  • Yukio Fukuyama
  • Georg F Hoffmann
  • Karin Jurkat-Rott
  • Anna-Kaisa Anttonen
  • Gerhard Kurlemann
  • Anna-Elina Lehesjoki
  • Frank Lehmann-Horn
  • Massimo Mastrangelo
  • Ulrike Mause
  • Stephan Müller
  • Bernd Neubauer
  • Burkhard Püst
  • Dietz Rating
  • Angela Robbiano
  • Susanne Ruf
  • Christopher Schroeder
  • Andreas Seidel
  • Nicola Specchio
  • Ulrich Stephani
  • Pasquale Striano
  • Jens Teichler
  • Dilsad Turkdogan
  • Federico Vigevano
  • Maurizio Viri
  • Peter Bauer
  • Federico Zara
  • Holger Lerche
  • Yvonne G Weber
چکیده

Mutations in PRRT2 have been described in paroxysmal kinesigenic dyskinesia (PKD) and infantile convulsions with choreoathetosis (PKD with infantile seizures), and recently also in some families with benign familial infantile seizures (BFIS) alone. We analyzed PRRT2 in 49 families and three sporadic cases with BFIS only of Italian, German, Turkish, and Japanese origin and identified the previously described mutation c.649dupC in an unstable series of nine cytosines to occur in 39 of our families and one sporadic case (77% of index cases). Furthermore, three novel mutations were found in three other families, whereas 17% of our index cases did not show PRRT2 mutations, including a large family with late-onset BFIS and febrile seizures. Our study further establishes PRRT2 as the major gene for BFIS alone.

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منابع مشابه

The PRRT2 mutation c.649dupC is the so far most frequent cause of benign familial infantile convulsions

PURPOSE Mutations in the PRRT2 gene have been recently described as a cause of paroxysmal kinesigenic dyskinesia, infantile convulsions with choreoathetosis syndrome and, less often, infantile convulsions. We have analysed the frequency of PRRT2 mutations in families with benign familial infantile convulsions without paroxysmal kinesigenic dyskinesia. METHODS AND RESULTS Direct sequencing of ...

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Genetic analysis of PRRT2 for benign infantile epilepsy, infantile convulsions with choreoathetosis syndrome, and benign convulsions with mild gastroenteritis.

PURPOSE PRRT2 mutations were recently identified in benign familial infantile epilepsy (BFIE) and infantile convulsions with paroxysmal choreoathetosis (ICCA) but no abnormalities have so far been identified in their phenotypically similar seizure disorder of benign convulsions with mild gastroenteritis (CwG), while mutations in KCNQ2 and KCNQ3 have been recognized in benign familial neonatal e...

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Phenotypes and PRRT2 mutations in Chinese families with benign familial infantile epilepsy and infantile convulsions with paroxysmal choreoathetosis

BACKGROUND Mutations in the PRRT2 gene have been identified as the major cause of benign familial infantile epilepsy (BFIE), paroxysmal kinesigenic dyskinesia (PKD) and infantile convulsions with paroxysmal choreoathetosis/dyskinesias (ICCA). Here, we analyzed the phenotypes and PRRT2 mutations in Chinese families with BFIE and ICCA. METHODS Clinical data were collected from 22 families with ...

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PRRT2 Mutations Are Related to Febrile Seizures in Epileptic Patients

Previous studies reported that the proline-rich transmembrane protein 2 (PRRT2) gene was identified to be related to paroxysmal kinesigenic dyskinesia (PKD), infantile convulsions with PKD, PKD with migraine and benign familial infantile epilepsy (BFIE). The present study explores whether the PRRT2 mutation is a potential cause of febrile seizures, including febrile seizures plus (FS+), general...

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Next-generation sequencing has identified mutations in the PRRT2 (proline-rich transmembrane protein 2) gene as the leading cause for a wide and yet evolving spectrum of paroxysmal diseases. PRRT2 mutations are found in the majority of patients with benign familial infantile epilepsy, infantile convulsions and choreoathetosis and paroxysmal kinesigenic dyskinesia, confirming a common disease sp...

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عنوان ژورنال:
  • Human mutation

دوره 33 10  شماره 

صفحات  -

تاریخ انتشار 2012